Kock pouch
Neobladder-to-urethra diversion
Florida pouch
Indiana pouch
A. Kock pouch
dissolving homogenous nucleation
high tendency to form cystine sulfate stones
indicates the amount of dietary protein
post ESWL therapy
chemical analysis of a recovered stone
hypercalcemia
CT finding
high breakability on ESWL
the commonest to form staghorn giant calculi
formed by urease producing bacteria
antibiotics have a role in the treatment
form at the two extremes of urinary pH range
patients should be given large amounts of fluids to hasten stones passage
fluids are given to keep the patient well hydrated
the recommended regimen is 2 L of ringer lactate over 2 hours
fluids are contraindicated if desmopressin (DDAVP) was given
furosemide
tamsulosin
nifedipine
diclofenac
frequently caused by loop diuretics
stones are often radiolucent
may be reversed by the use of thiazides
low calcium-to-creatinine ratio predicts stones resolution
Ca.oxalate, Ca.phosphate, Na.urate, cystine
Ca.phosphate, Ca.oxalate, cystine, Na.urate
Ca.oxalate, Ca.phosphate, cystine, Na.urate
Ca.phosphate, Ca.oxalate, Na.urate, cystine
increase urinary calcium, oxalate, and uric acid excretion
decrease urinary calcium; but increase oxalate, and uric acid excretion
increased urinary calcium and uric acid; but decrease oxalate excretion
decreased urinary calcium, oxalate, and uric acid excretion
physiological saline 0.9%
glycine 1.5%
balanced salt solution
distilled water
spinal cord injury
senile enlargement of prostate
augmented bladder
neurogenic hyper-reflexive bladder
form due to inspissated smegma
form due to stasis of urinary salts
cause inguinal lymphadenopathy
often associated with phimosis in uncircumcised males
have diagnostic hexagonal crystals
dont respond to ESWL therapy
are highly soluble in water
inherited in an autosomal recessive fashion
aminoglycosides
macrolides
cephalosporins
fluoroquinolones
Ca.oxalate monohydrate
cystine
matrix
Ca.oxalate dihydrate
≥ 2 cm diameter
upper calyx location
Na.urate composition
600 - 800 HU density
upper, lower, mid
lower, upper, mid
mid, upper, lower
mid, lower, upper
stone size
unrelieved obstruction
infection and septicemia
recurrent stone formation
xanthine
ammonium urate
cystine
calcium oxalate dihydrate
best treated by total parathyroidectomy
first-time stone formers are at a 50% risk for recurrence
males have higher recurrence rate than females
stone formers produce stones of the same type every time
urate
triple phosphate
oxalate monohydrate
matrix
they are multiple and small in size
usually, they are voided spontaneously
they, rarely, form large stones within the peripheral zone
contrast CT is the conventional method for diagnosis
α-Mercaptopropionylglycine
d-Penicillamine
a & b
none of the above
wet chemical analysis
thermogravimetry
scanning electron microscopy
none of the above
stones in a solitary kidney
ureteral stones causing bilateral obstructions
a kidney stone of ≥ 2.5 cm in size
all of the above
commoner in females than in males
in pediatrics, are of calcium oxalate and/or ammonium urate composition
caused by bladder outlet obstruction
might result in bladder cancer
stone size of ≤ 4 mm
stone burden of ≥ 22 mm
there is a distal partial obstruction
the patient has end-stage renal failure
transitional epithelium lining minor calyces
transitional epithelium lining major calyces
basement membrane of the loops of Henle
papillary tips of polar pyramids
placement of a percutaneous nephrostomy drain
surgical exploration and primary repair
conservative management
endoscopic retrograde ureteral stenting
lithogenic anion to cation ratio
Randall cut off
saturation index
solubility product
by performing intra-operative ultrasonography
by performing radial nephrotomies
by performing adjunct PCLN
by taking a scout KUB film