smaller incisions with a shorter hospital stay
lower risk for blood transfusion
none of the above
. What is the first-line management of uncomplicated LUTS due to large prostate?
B. lower risk for blood transfusion
as a man ages, the responsiveness of prostate cells to androgenic stimuli decreases
adrenal androgens have no role in BPH development
type-1 steroid 5 α-reductase is functionally active in the hair follicle
all of the above
intra-prostatic levels of estrogen decrease in men with BPH
stimulation of the adrenergic nervous system results in a dynamic increase in prostatic urethral resistance
inflammation may play a role through cytokines to promote cell growth
hyperplasia occurs due to an imbalance between cell death and cell proliferation
the irrigating fluid is at a pressure exceeding 10 mm Hg
the prostate volume is > 45 cc
the resection time is > 90 minutes
all of the above
enoxaparin
silodosin
finasteride
tolterodine
before catheterization
after catheterization and before TURP
immediately after TURP
2 weeks after TURP
go for urine cytology testing
go for PSA testing
go for TRUS
use anticholinergic medication
2 weeks after performing prostatic urethral lift
6-month treatment with 5α-reductase inhibitors
after placing a stent in the prostatic urethra
immediately after removing one-half of the prostate by TURP
milder postoperative hematuria
tension-free bladder closure
extra-peritoneal approach
. When comparing TURP to open prostatectomy for removing prostatic adenoma, the latter has the following advantages, EXCEPT:
PSA decreases
the prostate size decreases
the complaints resolve
the Q.O.L improves
nearly all men have PVR of less than 12 ml
it predicts the outcome of surgical treatment
it correlates well with BPH/LUTS
it is diagnostic for bladder outlet obstruction
symptoms begin with a serum sodium of less than 120 mEq/L
the mortality is 2.7-5.8 %
manifestations rely on acute changes in the intravascular volume and plasma solute concentrations
the preferred height of irrigating fluid is 60 cm above the patient
increased intravesical pressure
increased detrusor pressure
increase collagen deposition in the detrusor
detrusor smooth muscle hypertrophy
never
unlikely
likely
always
bladder trabeculation
significant PVR
low peak flow rate
high IPSS
the glandular component of the prostate
the IPSS questionnaire points
the PVR
the transitional zone volume
hematuria
recurrent urinary tract infection
renal insufficiency
all of the above
damage to the internal sphincter
damage to the external sphincter
bladder perforation
damage to a ureteral orifice
worsening of LUTS and BPH over time
patients die of other reasons before serious complications occur
physically, the space of prostatic fossa limits the gland enlargement
ultimately, the gland will degenerate and undergo apoptosis
the presence of prostate cancer
previous prostatectomy
all of the above
. Robot-assisted laparoscopic prostatectomy for prostatic adenoma has the following advantage over TURP:
LUTS with neurologic disease
LUTS with post-void dribble
LUTS with suspicious DRE
LUTS with hematuria
tamsulosin
alfuzosin
doxazosin
silodosin
sizable bladder stones
Hutch diverticulum
a suspicion of cancer
a & b
the neurological status of the patient
PVR
severity of obstructive LUTS
all of the above
62 - 78%
48 - 61%
79 - 93%
34 - 47%
tamsulosin 0.8 mg
reassurance
repeat total and free PSA
diagnostic cystoscopy
it helps predict the response to 5α-reductase inhibitors
it monitors LUTS/BPH progression
BPH patients are at higher risk of developing prostate cancer
a & b
bladder neck
apex
median lobe
para-collecular
small prostate
mainly median lobe hypertrophy
history of urinary retention
mainly irritative symptoms
tuberculous prostatitis
prostatic cancer
inspissated prostatic abscess
any of the above
the most potent androgenic hormones in BPH development is DHT
type-2 steroid 5 α-reductase, is most commonly found in the prostate
castrated individuals before puberty will not develop BPH
as a man ages, the number of androgen receptors in the prostate decreases