it causes reduction in the mean intra-ureteral pressure
it reduces the pain of acute renal colic
it has a direct relaxing effect on the renal pelvis and ureteral musculature
it is indicated when stones are ≤ 4 mm in diameter
D. it is indicated when stones are ≤ 4 mm in diameter
should be treated with more analgesics
must undergo metabolic worked out
are unlikely to pass out spontaneously
chemolysis should be tried first
by lowering urinary saturation of Ca.oxalate
by preventing heterogeneous nucleation of Ca.oxalate
by inhibiting spontaneous precipitation and agglomeration of Ca.oxalate
by all of the above
spinal cord injury
senile enlargement of prostate
augmented bladder
neurogenic hyper-reflexive bladder
3 6 %
12 15 %
0.4 0.8 %
0.09 0.14 %
lithogenic anion to cation ratio
Randall cut off
saturation index
solubility product
to stent the ureter after ureteral surgery
to facilitate stone passage
after a tough ureteroscopy procedure
all of the above
short and wide infundibulum
large lower-pole infundibulo-pelvic angle
the adjunct usage of PCNL
all of the above
furosemide
tamsulosin
nifedipine
diclofenac
size of 5 mm
location at the lower calyx
density of 400 HU
being recurrent
urate
triple phosphate
oxalate monohydrate
matrix
ammonium urate
sodium urate
calcium oxalate
calcium phosphate
≥ 2 cm diameter
upper calyx location
Na.urate composition
600 - 800 HU density
2 days
2 weeks
2 months
4 6 hrs.
frequently caused by loop diuretics
stones are often radiolucent
may be reversed by the use of thiazides
low calcium-to-creatinine ratio predicts stones resolution
the preferred access into the collecting system is through a posterior calyx
the posterior calyceal group is typically more medial than in the normal kidney
in most cases the lower pole calyces are posterior
it is desirable to make an upper pole collecting system puncture
high citrate, high oxalate
low citrate, low oxalate
high citrate, low oxalate
low citrate, high oxalate
left ureterocele
bifid right renal pelvis
neurogenic bladder
bilateral UPJ stenosis
watchful waiting
open surgical correction
IVU with possible endoscopic ureteral stenting
perc. nephrostomy tube insertion
the initial step is papillary plaque formation
crystals formation occurs inside the nephron
tubular precipitates form harmless crystalluria
the attraction of organic compounds and activation crystallization is regulated by osteopontin
piezoelectric
electrohydraulic
electromagnetic
microexplosive
because urine and body secretions are highly concentrated
due to renal leak hypercalciuria
as a result of distal renal tubular acidosis type I
because of reduced or absent of oxalobacter formigenes colonization
limit beef, chicken, pork, eggs, fish, shellfish, and other animal proteins
limit beans, nuts, chocolate, coffee, dark green vegetables, and soda
limit canned, packaged, and fast foods
limit milk, cheese, and other dairy products
stone size
unrelieved obstruction
infection and septicemia
recurrent stone formation
orthophosphates may have a role in the treatment
hyperparathyroidectomy and levothyroxine replacement is the optimum treatment
management includes Calcium chelating agent and repeat 24hr urine collection in 3 months
surgical excision of the adenoma(s) is the treatment of choice
stone chemical composition
stone burden
first stone vs. recurrent
stone density
can be forgotten in place
vesico-renal reflux
calyceal perforation
detrusor irritability and/or hematuria
they are mandatory when urine shows ≥ 10 WBCs/hpf in symptomatic patients
they aim at treating pyonephrosis and urosepsis
they should cover Escherichia coli and Staphylococcus, Enterobacter, Proteus, and Klebsiella species
All of the above
composed of calcium phosphate and calcium carbonate
the vast majority are asymptomatic
most of the calculi are found in the transitional zone
they dont affect PSA levels
right hemicolectomy
small bowel resection
Roux-en-Y gastric bypass
b & c
upper, lower, mid
lower, upper, mid
mid, upper, lower
mid, lower, upper