orthophosphates may have a role in the treatment
hyperparathyroidectomy and levothyroxine replacement is the optimum treatment
management includes Calcium chelating agent and repeat 24hr urine collection in 3 months
surgical excision of the adenoma(s) is the treatment of choice
D. surgical excision of the adenoma(s) is the treatment of choice
3 6 %
12 15 %
0.4 0.8 %
0.09 0.14 %
a stone in the lower calyx with a wide mouth of infundibulum and obtuse lower calyx to ureter angle
a stone in an anterior group calyceal diverticulum with thin overlying renal parenchyma
5 mm calcium-containing stone in an intrarenal pelvis and wide UPJ
6 years post anatrophic nephrolithotomy, recurrent mid calyceal stone
size of 5 mm
location at the lower calyx
density of 400 HU
being recurrent
localization of stones in the ureter is difficult or impossible
inability to visualize stones breaking down in real time
c. patient`s position on ESWL table is uncomfortable
d. inability to visualize radiolucent stones
ammonium urate
sodium urate
calcium oxalate
calcium phosphate
calcium monohydrate
calcium oxalate
ammonium urate
none of the above
indinavir
magnesium ammonium phosphate
xanthine
matrix
ESWL
URS
PCNL
none of the above
the initial step is papillary plaque formation
crystals formation occurs inside the nephron
tubular precipitates form harmless crystalluria
the attraction of organic compounds and activation crystallization is regulated by osteopontin
stone chemical composition
stone burden
first stone vs. recurrent
stone density
left ureterocele
bifid right renal pelvis
neurogenic bladder
bilateral UPJ stenosis
composed of calcium phosphate and calcium carbonate
the vast majority are asymptomatic
most of the calculi are found in the transitional zone
they dont affect PSA levels
urate
triple phosphate
oxalate monohydrate
matrix
by lowering urinary saturation of Ca.oxalate
by preventing heterogeneous nucleation of Ca.oxalate
by inhibiting spontaneous precipitation and agglomeration of Ca.oxalate
by all of the above
orthophosphates may have a role in the treatment
hyperparathyroidectomy and levothyroxine replacement is the optimum treatment
management includes Calcium chelating agent and repeat 24hr urine collection in 3 months
surgical excision of the adenoma(s) is the treatment of choice
can be forgotten in place
vesico-renal reflux
calyceal perforation
detrusor irritability and/or hematuria
400 600 HU
600 800 HU
800 1000 HU
1000 1200 HU
α-Mercaptopropionylglycine
acetohydroxamic acid
carbonic anhydrase inhibitors
xanthine oxidase inhibitors
terminal ilium resection
liver transplantation
kidney transplantation
regular hemodialysis
placing the patient in anti-Trendelenburg position
making the puncture under local anesthesia
injection Co2 gas to create a safety space under the diaphragm before puncturing
making the puncture during full expiration
active UTI is an absolute contraindication
fluoroquinolone is the first choice for antimicrobial prophylaxis
withholding aspirin for only 10 days is enough
despite sterile urine, stone fragmentation might release hidden bacterial endotoxins and viable bacteria
hypercalciuria and hypocitraturia
hypercalciuria and hypercitraturia
hypocalciuria and hypocitraturia
hypocalciuria and hypercitraturia
blood cells more than pus cells
pus cells if infection was superadded
crystals might appear
all of the above
commonly unilateral
commonly due to repeated infections
urate stones are the second most common cause of staghorn calculi
ESWL monotherapy with ureteral stenting is the ideal treatment
to stent the ureter after ureteral surgery
to facilitate stone passage
after a tough ureteroscopy procedure
all of the above
placement of a percutaneous nephrostomy drain
surgical exploration and primary repair
conservative management
endoscopic retrograde ureteral stenting
0 10%
10 20%
20 30%
30 40%
1000 - 1300 HU density
5 - 10 mm diameter
lower calyx location
mid ureteral location
they are metabolic stones that form at high urinary pH
they score 800 1000 HU on CT
only 25% of affected patients have Gout disease
affected patients must stop eating animal protein
spinal cord injury
senile enlargement of prostate
augmented bladder
neurogenic hyper-reflexive bladder