400 600 HU
600 800 HU
800 1000 HU
1000 1200 HU
A. 400 600 HU
ammonium urate
sodium urate
calcium oxalate
calcium phosphate
Kock pouch
Neobladder-to-urethra diversion
Florida pouch
Indiana pouch
2 days
2 weeks
2 months
4 6 hrs.
by performing intra-operative ultrasonography
by performing radial nephrotomies
by performing adjunct PCLN
by taking a scout KUB film
composed of calcium phosphate and calcium carbonate
the vast majority are asymptomatic
most of the calculi are found in the transitional zone
they dont affect PSA levels
≥ 2 cm diameter
upper calyx location
Na.urate composition
600 - 800 HU density
impaired renal tubular calcium reabsorption
excessive glomerular leak of calcium
deficiency of the enzyme xanthine oxidase
hypercalcemia
the commonest to form staghorn giant calculi
formed by urease producing bacteria
antibiotics have a role in the treatment
form at the two extremes of urinary pH range
cystine
brushite
Ca.oxalate monohydrate
Ca.oxalate dihydrate
3 6 %
12 15 %
0.4 0.8 %
0.09 0.14 %
Tumor lysis syndrome
hypoparathyroidism
myeloproliferative disorder
Lesch-Nyhan syndrome
they are multiple and small in size
usually, they are voided spontaneously
they, rarely, form large stones within the peripheral zone
contrast CT is the conventional method for diagnosis
steinstrasse
a stone in ureterocele
fever, leucocytosis, pain
brushite stones
physiological saline 0.9%
glycine 1.5%
balanced salt solution
distilled water
high citrate, high oxalate
low citrate, low oxalate
high citrate, low oxalate
low citrate, high oxalate
indinavir
magnesium ammonium phosphate
xanthine
matrix
obese patients have a higher tendency for uric acid stone formation
high-protein, low-carbohydrate diet might increase the risk of stone formation and bone loss
metabolic syndrome is associated with high urinary pH
Roux-en-Y-gastric bypass surgery may increase the risk for stone formation
calcium phosphate
calcium oxalate monohydrate
sodium urate
2,8 dihydroxyadenine
have diagnostic hexagonal crystals
dont respond to ESWL therapy
are highly soluble in water
inherited in an autosomal recessive fashion
chemical analysis of a recovered stone
hypercalcemia
CT finding
high breakability on ESWL
right hemicolectomy
small bowel resection
Roux-en-Y gastric bypass
b & c
dissolves cystine stones
enhances nephrocalcinosis process over old scared areas
inhibits Ca.oxalate aggregation and crystallization
plays a secondary role in metastatic calcification process
the initial step is papillary plaque formation
crystals formation occurs inside the nephron
tubular precipitates form harmless crystalluria
the attraction of organic compounds and activation crystallization is regulated by osteopontin
result from an inherited defect of renal tubular reabsorption of cysteine
characteristically, urate and cysteine stone are radiolucent
cysteine is a dibasic amino acid
on plain X-ray, cysteine stones exhibit ground-glass appearance
dehydration
metabolic disorders
congenital anomalies
all of the above
because urine and body secretions are highly concentrated
due to renal leak hypercalciuria
as a result of distal renal tubular acidosis type I
because of reduced or absent of oxalobacter formigenes colonization
it is characterized by low urinary magnesium and citrate
magnesium increases renal tubular citrate resorption
diarrheal is a remarkable side effect of magnesium therapy
potassium-magnesium preparations might restore urinary magnesium and citrate levels
orthophosphates may have a role in the treatment
hyperparathyroidectomy and levothyroxine replacement is the optimum treatment
management includes Calcium chelating agent and repeat 24hr urine collection in 3 months
surgical excision of the adenoma(s) is the treatment of choice
ESWL
PCNL
radial nephrolithotomy
pyelolithotomy with ureteral stenting
commonly occur in patients with senile prostatic enlargement
common in children exposed to low-protein, low-phosphate diet
rarely recur after treatment
respond to ESWL