short skin-to-stone distance (SSD)
end-stage renal failure
a stone in the upper calyx
the presence of a 30 cm, 4.7 Fr ureteral stent in situ
B. end-stage renal failure
they are mandatory when urine shows ≥ 10 WBCs/hpf in symptomatic patients
they aim at treating pyonephrosis and urosepsis
they should cover Escherichia coli and Staphylococcus, Enterobacter, Proteus, and Klebsiella species
All of the above
form due to inspissated smegma
form due to stasis of urinary salts
cause inguinal lymphadenopathy
often associated with phimosis in uncircumcised males
α-Mercaptopropionylglycine
acetohydroxamic acid
carbonic anhydrase inhibitors
xanthine oxidase inhibitors
cysteine
urate
calcium
none of the above
400 600 HU
600 800 HU
800 1000 HU
1000 1200 HU
they are metabolic stones that form at high urinary pH
they score 800 1000 HU on CT
only 25% of affected patients have Gout disease
affected patients must stop eating animal protein
indinavir
magnesium ammonium phosphate
xanthine
matrix
stone size
unrelieved obstruction
infection and septicemia
recurrent stone formation
can be introduced from the bladder or kidney or any part of the ureter`s course
the standard adult size is 32 cm long, 4 mm calibre
might result in encrustations and ureteral obstruction
might result in ureteral dilation
they are multiple and small in size
usually, they are voided spontaneously
they, rarely, form large stones within the peripheral zone
contrast CT is the conventional method for diagnosis
α-Mercaptopropionylglycine
d-Penicillamine
a & b
none of the above
ESWL
URS
PCNL
none of the above
physiological saline 0.9%
glycine 1.5%
balanced salt solution
distilled water
cystine
brushite
Ca.oxalate monohydrate
Ca.oxalate dihydrate
renal stones are found in 20% of patients with primary HPT
acidic arrest promotes crystallisation of calcium phosphate stones related to HPT
HPT, vitamin D excess, and malignancy could lead to hypercalcemia and hypercalciuria
only surgery can cure primary HPT
xanthine
ammonium urate
cystine
calcium oxalate dihydrate
furosemide
tamsulosin
nifedipine
diclofenac
spinal cord injury
senile enlargement of prostate
augmented bladder
neurogenic hyper-reflexive bladder
dissolves cystine stones
enhances nephrocalcinosis process over old scared areas
inhibits Ca.oxalate aggregation and crystallization
plays a secondary role in metastatic calcification process
best treated by total parathyroidectomy
first-time stone formers are at a 50% risk for recurrence
males have higher recurrence rate than females
stone formers produce stones of the same type every time
orthophosphates may have a role in the treatment
hyperparathyroidectomy and levothyroxine replacement is the optimum treatment
management includes Calcium chelating agent and repeat 24hr urine collection in 3 months
surgical excision of the adenoma(s) is the treatment of choice
ciprofloxacin
indinavir
thiazides
triamterene
watchful waiting
open surgical correction
IVU with possible endoscopic ureteral stenting
perc. nephrostomy tube insertion
Proteus mirabilis
E. coli
Pseudomonas aeruginosa
Staphylococcus epidermidis
Ca.oxalate monohydrate
cystine
matrix
Ca.oxalate dihydrate
Klebsiella pneumonia
Morganella morganii
Proteus mirabilis
all of the above
localization of stones in the ureter is difficult or impossible
inability to visualize stones breaking down in real time
c. patient`s position on ESWL table is uncomfortable
d. inability to visualize radiolucent stones
3 6 %
12 15 %
0.4 0.8 %
0.09 0.14 %
right hemicolectomy
small bowel resection
Roux-en-Y gastric bypass
b & c
stones at posterior urethra could be pushed back to the bladder
stones at anterior urethra have to undergo a trial of milking out, using copious intra-urethral xylocaine gel
often respond to a two-week course of tamsulosin
respond to Holmium laser treatment