xanthine
ammonium urate
cystine
calcium oxalate dihydrate
C. cystine
can be forgotten in place
vesico-renal reflux
calyceal perforation
detrusor irritability and/or hematuria
size of 5 mm
location at the lower calyx
density of 400 HU
being recurrent
active UTI is an absolute contraindication
fluoroquinolone is the first choice for antimicrobial prophylaxis
withholding aspirin for only 10 days is enough
despite sterile urine, stone fragmentation might release hidden bacterial endotoxins and viable bacteria
urate
triple phosphate
oxalate monohydrate
matrix
they are mandatory when urine shows ≥ 10 WBCs/hpf in symptomatic patients
they aim at treating pyonephrosis and urosepsis
they should cover Escherichia coli and Staphylococcus, Enterobacter, Proteus, and Klebsiella species
All of the above
cystine
brushite
Ca.oxalate monohydrate
Ca.oxalate dihydrate
lithogenic anion to cation ratio
Randall cut off
saturation index
solubility product
because urine and body secretions are highly concentrated
due to renal leak hypercalciuria
as a result of distal renal tubular acidosis type I
because of reduced or absent of oxalobacter formigenes colonization
commonly occur in patients with senile prostatic enlargement
common in children exposed to low-protein, low-phosphate diet
rarely recur after treatment
respond to ESWL
steinstrasse
a stone in ureterocele
fever, leucocytosis, pain
brushite stones
dissolving homogenous nucleation
high tendency to form cystine sulfate stones
indicates the amount of dietary protein
post ESWL therapy
chemical analysis of a recovered stone
hypercalcemia
CT finding
high breakability on ESWL
they are multiple and small in size
usually, they are voided spontaneously
they, rarely, form large stones within the peripheral zone
contrast CT is the conventional method for diagnosis
have diagnostic hexagonal crystals
dont respond to ESWL therapy
are highly soluble in water
inherited in an autosomal recessive fashion
right hemicolectomy
small bowel resection
Roux-en-Y gastric bypass
b & c
they are metabolic stones that form at high urinary pH
they score 800 1000 HU on CT
only 25% of affected patients have Gout disease
affected patients must stop eating animal protein
ciprofloxacin
indinavir
thiazides
triamterene
by performing intra-operative ultrasonography
by performing radial nephrotomies
by performing adjunct PCLN
by taking a scout KUB film
left ureterocele
bifid right renal pelvis
neurogenic bladder
bilateral UPJ stenosis
2,8 dihydroxyadenine stones
sulfa medications-induced stones
calcium oxalate stones
matrix stones
subcostal puncture performed during full expiration
previous open nephrolithotomy
access lateral to the posterior axillary line
horseshoe kidney
can be placed through perc. nephrostomy
might slip out, especially in females
usually radiopaque
all of the above
blood cells more than pus cells
pus cells if infection was superadded
crystals might appear
all of the above
uncontrollable bleeding
incomplete removal of stones
pneumothorax
colonic perforation
the energy density of the shock waves as they pass through the skin
the size of the focal point
a & b
none of the above
Proteus mirabilis
E. coli
Pseudomonas aeruginosa
Staphylococcus epidermidis
α-Mercaptopropionylglycine
d-Penicillamine
a & b
none of the above
hypertensive patient
patient on aspirin withheld 5 days prior to ESWL
a stone in a scared poorly functioning kidney
ESWL every other day
matrix
indinavir
brushite
2,8 dihydroxyadenine
stone size
unrelieved obstruction
infection and septicemia
recurrent stone formation