aminoglycoside
fluoroquinolone
2nd generation cephalosporin
doxycycline
B. fluoroquinolone
16
18
22
12
rarely, the urothelial cell nests show a central lumen lined by glandular epithelium
In some cases, it may form polypoid masses that mimic urothelial neoplasms
It might appear as multinodular exophytic mass seen on cystoscopy
cystitis cystica and cystitis glandularis frequently coexist in the same specimen
N. gonorrhea and C. trachomatis
E. coli and Pseudomonas species
Mycoplasma genitalium and Ureaplasma species
Trichomonas vaginalis and Gardnerella vaginalis
are premalignant, and found in 1-6% of prostate biopsies
are small hyaline masses of unknown significance found in the prostate gland
they are degenerate cells or thickened secretions in the prostate ducts
might appear as prostate calcifications on X-ray KUB
P blood group
fimbria
pili
hemolysin
type I could harbor prostate abscess
type II presents as intermittent urinary tract infections
type III-a presentation might include psychological complaints
between 10-15% of men with type IV, have pus cells in their semen but no symptoms
is an uncommon granulomatous disease that affect the skin and/or urinary bladder
it might be due to a disturbed function of B lymphocytes
characterized by the presence of basophilic inclusion structure (Michaelis-Gutmann body)
it might be due to a defective phagolysosomal activity of monocytes or macrophages
sterile pyuria on 3 consecutive cultures
the presence of glomerulations and/or Hunner`s ulcer on endoscopy
pain and discomfort related to the bladder
urgency and frequency with no documented infection
giggle incontinence
estrogen deficiency
cystitis glandularis
cystitis cystica
produces yellow whitish, scanty, frothy urethral discharge
shows gram (+), extracellular diplococcic
infection could be contracted from the spouses eyes
responds fairly to azithromycin
nucleic acid amplification tests are the preferred to diagnose Gonococcal and Chlamydial infections
shows dark yellow, purulent, thick urethral discharge
the most common site of the infection is the endocervix
the incubation period is 2 3 weeks
could result from intra-renal abscess of ascending infection
urine culture might be negative
plain KUB X-ray has no value in the diagnosis
surgical drainage is the proper treatment
CT shows the characteristic bear paw sign
it is an infected, obstructed, poorly functioning kidney containing stones
nephrectomy is the treatment
all of the above
antibiotic therapy is recommended for affected individuals with documented trichomonal infection and sexual partners even if asymptomatic
empirical treatment for gonococcal urethritis should cover chlamydia trachomatis
consistent and proper usage of condoms is estimated to prevent HIV transmission by approximately 80 to 95%
vaccinations are available for the prevention of human papillomavirus, N. gonorrhea, chlamydia trachomatis
probably due to antibody/antigen reaction
has no diagnostic findings on cystoscopy
has no specific medical therapy
on histology, Von Brunn`s nests appear invaginating the urothelium into the lamina propria
type II
type III-a
type III-b
type IV
kidneys
bladder
prostate
epididymis
no pathognomonic histology for interstitial cystitis
basically, biopsies are performed to exclude carcinomas and other varieties of cystitis
diagnostic biopsies include the presence of discrete micro-ulcers and increased numbers of mast cells in the detrusor muscle or submucosa
none of the above
aminopenicillins
fluoroquinolones
aminoglycosides
nitrofurantoins
commonly, TB enters the urinary tract via intravesical instillation of attenuated live BCG to treat bladder cancer
CT urography may show infundibular stricture with or without hydrocalicosis
renal ultrasonography reveals calyceal erosions moth-eaten calyx
TB of the vas appears, clinically, as a thin hard strictured tube
is best diagnosed by ascending urethrography
occurs mostly in diabetic and immunosuppressed patients
could be due to maceration injury, irritant dermatitis, or Candida
commonly presents with deep inguinal lymphadenopathy
categorizes CP-CPPS, IC, and painful bladder syndrome based on 5 etiological principles
meant to classify CP-CPPS and IC patients into 6 domains
helps establish a reliable diagnosis of CP/CPPS or IC
the diagnostic scores of UPOINT depend on cystoscopy, TRUS, urine analysis and culture of uncommon microbes
might rupture into the collecting system causing (hydatiduria)and renal colic
are formed by the eggs of the tapeworm Echinococcus granulosus
most cysts are asymptomatic but might manifest as flank mass, dull pain, or hematuria
the most reliable diagnostic test uses partially purified hydatid arc 5 antigens in a double-diffusion test
inhibits bladder epithelial cell proliferation
inhibits the bladder proliferative growth factors
stimulates the proliferation inhibitory factors
none of the above
type I
type II
type III
type IV
30
40
50
60
pH of vaginal secretions increases after menopause
estrogen deficiency manifests as trophic urethritis and atrophic vaginitis
topical conjugated estrogen replacement carries a significant risk of breast and endometrial cancers
manifestations might include obstructive symptoms and non-infectious cystitis
kidneys, prostate and epididymi
bladder, ureters and renal pelvis
vasa, scrotum and adrenals
testes, bladder neck and seminal vesicles
natural sloughing of bladder mucosa
voiding
urine osmolarity
urine pH
an esinophilic immune reaction is generated in response to the eggs
chronic schistosomiasis can eventually result in small bladder and the development of cancers
schistosoma mansoni often causes urinary tract infections
could cause inflammatory polys and recurrent hematuria